Belly Fat Causes

Belly fat causes - the evolution of visceral fat

Belly Fat Causes – Check your Lifestyle for Clues

To lose unhealthy visceral belly fat, it is important to realise where it came from. We must also consider that although our bodies are designed to function in the same way, our sex, unique genetics and lifestyle factors play a massive role in how our fat is stored throughout our lifetime.

Common Causes of Belly Fat

Unhealthy Eating Habits - Wrong Food, Wrong Time of Day, Lack of Nutrients
Muscle nutrition offers insight into healthy eating (Read muscle science and nutrition for tips)

Sleeplessness and Insomnia - Both the amount and the quality of sleep we get each night is of great importance. Sleep is a period in the 24 hr circadian cycle that is where we do our best maintenance on the body, right down to the cellular level. Delta waves are present in deep sleep and GH peaks in this environment, offering a highly anabolic arena from which to benefit. Sleep loss, particularly that which occurs regularly, has the ability to promote hormonal dysfunction. Weight gain is often the result of sleep deprivation., the amount and quality of sleep we get each night is important to the maintenance of our health.

Chronic Stress – High Cortisol Levels  – Stress levels are high in today’s world. The number of roles we play, the number of chemicals we have exposure to, our bodies are under constant attack with cortisol levels being elevated consistently. Chronic stress has been linked to weight gain. We need to learn how to reduce high cortisol levels so our health does not suffer the consequences of hormonal dysregulation.

Inactivity – Lack of Physical Exercise and Activity
As we grow older, we often become better-educated regarding the right way to exercise in order to maintain muscle mass and vitality. Studies have revealed that the intensity of our training regime is of the utmost importance in maintaining a healthy weight. We must support muscle hypertrophy by keeping in mind, what our muscle cells need to grow bigger and stronger. 

It is safe to say that by the age of 30 we should take an active interest in our hormonal levels and get them checked regularly. As you will come to find out, it is extremely important to acknowledge the power of the endocrine system, our hormones and keeping these levels within a healthy balance.

Understanding belly fat causes, (essentially what mistakes you have been making) and the dangers that present themselves when belly fat (visceral fat) is covering vital organs will be discussed.
The Dangers of Visceral Fat

How to Lose Belly Fat

The Types of Fat (The Unsightly and the Deadly)

Subcutaneous Fat

Location: Directly Under Skin
Subcutaneous fat is that which you can see and pinch, which is located directly under the skin. It is soft when you poke at it and though we don’t like wearing it, subcutaneous fat is not the dangerous fat that leads to disease and death.

Visceral Fat

Location: Abdominal Region, Belly Fat, Surrounds and Blankets Vital Organs
The fat you should worry about is visceral fat (belly fat). Visceral fat is stored around your abdominal cavity, blanketing organs such as the pancreas, kidneys, liver and intestines. It is that which makes your belly feel firm and is of a gel-like consistency.

Belly Fat Causes

The increase of belly fat (visceral fat storage) may be a result of:

Belly Fat (Visceral Fat) is Dangerous and Can Cause Death!

  • Fat cells are known to act independently as an organ pumping out hormones and inflammatory substances called Cytokines.
  • Cytokines interfere with hormones that regulate one’s appetite, weight, mood and even plays havoc on brain function.
  • Cytokines may act on the very cells that are responsible for secreting them (autocrine action).
  • Cytokines may also act on nearby cells which is referred to as a paracrine action.
  • Sometimes cytokines act on distant cells (endocrine action).

The danger of belly fat is that it leads to suffering a reduced quality of life and ultimately death:

Insulin Resistance and Visceral Fat Storage
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    Proc Soc Exp Biol Med. 1992 Jun;200(2):197-201. No abstract available.
  • Visceral Fat Volume is a Better Predictor for Insulin Resistance than Abdominal Wall Fat Index in Patients with Prediabetes and Type 2 Diabetes Mellitus
    Ozlem Ozer Cakir1*, Mehmet Yildiz2 and Mustafa Kulaksizoglu3
  • Su Youn Nam,1,2,* Il Ju Choi,3,* Kum Hei Ryu,1 Bum Joon Park,1 Young-Woo Kim,3 Hyun Beom Kim,4 and Jeongseon Kim5
    The Effect of Abdominal Visceral Fat, Circulating Inflammatory Cytokines, and Leptin Levels on Reflux Esophagitis
    J Neurogastroenterol Motil. 2015 Apr; 21(2): 247–254.
  • Debette, StĂ©phanie et al. “Visceral Fat Is Associated with Lower Brain Volume in Healthy Middle-Aged Adults.” Annals of neurology 68.2 (2010): 136–144. PMC. Web. 8 Aug. 2016.
  • Giles, Jon T. et al. “Abdominal Adiposity in Rheumatoid Arthritis: Association with Cardiometabolic Risk Factors and Disease Characteristics.” Arthritis and rheumatism 62.11 (2010): 3173–3182. PMC. Web. 8 Aug. 2016.
  • Visceral Adiposity, Insulin Resistance, and Type 2 Diabetes
    American Journal of Lifestyle Medicine May/June 2010 4: 230-243, first published on March 2, 2010
  • Donohoe, Claire L, Suzanne L Doyle, and John V Reynolds. “Visceral Adiposity, Insulin Resistance and Cancer Risk.” Diabetology & Metabolic Syndrome 3 (2011): 12. PMC. Web. 8 Aug. 2016.
  • Visceral Fat Adipokine Secretion Is Associated With Systemic Inflammation in Obese Humans
    Crossref DOI link:
    Published: 2007-04-01
  • Tijdschr Psychiatr. 2011;53(9):613-20.
    [Depressive symptoms, cortisol, visceral fat and metabolic syndrome].
  • Guedes, Erika P et al. “Body Composition and Depressive/anxiety Symptoms in Overweight and Obese Individuals with Metabolic Syndrome.” Diabetology & Metabolic Syndrome 5 (2013): 82. PMC. Web. 8 Aug. 2016.
    Low Testosterone Associated With Obesity and the Metabolic Syndrome Contributes to Sexual Dysfunction and Cardiovascular Disease Risk in Men With Type 2 Diabetes
    Christina Wang, MD1⇓, Graham Jackson, MD2, T. Hugh Jones, MD3, Alvin M. Matsumoto, MD4, Ajay Nehra, MD5, Michael A. Perelman, PHD6, Ronald S. Swerdloff, MD1, Abdul Traish, PHD7, Michael Zitzmann, MD8 and Glenn Cunningham, MD9